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A novel DSPP mutation causes dentinogenesis imperfecta type II in a large Mongolian family.
[dentinogenesis imperfecta]
Several
studies
have
shown
that
the
clinical
phenotypes
of
dentinogenesis
imperfecta
type
II
(
DGI
-
II
)
may
be
caused
by
mutations
in
dentin
sialophosphoprotein
(
DSPP
)
.
However
,
no
previous
studies
have
documented
the
clinical
phenotype
and
genetic
basis
of
DGI
-
II
in
a
Mongolian
family
from
China
.
We
identified
a
large
five
-generation
Mongolian
family
from
China
with
DGI
-
II
,
comprising
64
living
family
members
of
whom
22
were
affected
.
Linkage
analysis
of
five
polymorphic
markers
flanking
DSPP
gene
was
used
to
genotype
the
families
and
to
construct
the
haplotypes
of
these
families
.
All
five
DSPP
exons
including
the
intron-exon
boundaries
were
PCR-amplified
and
sequenced
in
48
members
of
this
large
family
.
All
affected
individuals
showed
discoloration
and
severe
attrition
of
their
teeth
,
with
obliterated
pulp
chambers
and
without
progressive
high
frequency
hearing
loss
or
skeletal
abnormalities
.
No
recombination
was
found
at
five
polymorphic
markers
flanking
DSPP
in
the
family
.
Direct
DNA
sequencing
identified
a
novel
A--
>
G
transition
mutation
adjacent
to
the
donor
splicing
site
within
intron
3
in
all
affected
individuals
but
not
in
the
unaffected
family
members
and
50
unrelated
Mongolian
individuals
.
This
study
identified
a
novel
mutation
(
IVS
3
+
3
A--
>
G
)
in
DSPP
,
which
caused
DGI
-
II
in
a
large
Mongolian
family
.
This
expands
the
spectrum
of
mutations
leading
to
DGI
-
II
.
Diseases
Validation
Diseases presenting
"severe attrition"
symptom
dentin dysplasia
dentinogenesis imperfecta
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