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Subthreshold psychotic symptoms in 22q11.2 deletion syndrome.
[22q11.2 deletion syndrome]
Chromosome
22
q
11
.
2
deletion
syndrome
(
22
q
11
DS
)
confers
25
%
risk
for
psychosis
and
is
an
invaluable
window
for
understanding
the
neurobiological
substrate
of
psychosis
risk
.
The
Structured
Interview
for
Prodromal
Syndromes
(
SIPS
)
is
well
validated
in
nondeleted
populations
for
detecting
clinical
risk
but
has
only
recently
been
applied
to
22
q
11
DS
.
We
assessed
the
largest
22
q
11
DS
cohort
to
date
and
report
on
SIPS
implementation
and
symptoms
elicited
.
The
SIPS
,
including
its
19
subscales
,
was
administered
to
157
individuals
with
22
q
11
DS
aged
8
to
25
years
.
Youth
and
caregiver
interviews
were
conducted
and
rated
separately
,
then
compared
for
agreement
.
Implementation
of
the
SIPS
in
22
q
11
DS
was
challenging
because
of
the
prevalence
of
developmental
delay
and
comorbid
conditions
.
However
,
by
explaining
questions
and
eliciting
examples
,
we
were
able
to
help
youths
and
caregivers
understand
and
respond
appropriately
.
Consensus
ratings
were
formulated
and
analyzed
with
itemwise
and
factor
analysis
.
Subthreshold
symptoms
were
common
,
with
85
%
of
individuals
endorsing
1
or
more
.
The
most
commonly
rated
items
were
ideational
richness
(
47
%
)
and
trouble
with
focus
and
attention
(
44
%
)
.
Factor
analysis
revealed
a
3
-
factor
solution
with
positive
,
negative
,
and
disorganized
components
.
Youth-caregiver
comparisons
suggested
that
youths
report
greater
symptoms
of
perceptual
abnormalities
,
suspiciousness
,
trouble
with
emotional
expression
,
and
bizarre
thinking
.
Caregivers
reported
more
impaired
tolerance
to
normal
stress
,
poor
hygiene
,
and
inattention
.
The
SIPS
was
adapted
for
22
q
11
DS
through
comprehensive
and
semi-structured
administration
methods
,
yielding
a
high
prevalence
of
subthreshold
psychotic
symptoms
.
The
significance
and
predictive
validity
of
these
symptoms
require
future
longitudinal
analysis
.
Diseases
Validation
Diseases presenting
"is an invaluable window for understanding the neurobiological substrate of psychosis risk"
symptom
22q11.2 deletion syndrome
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